Sprinkled in with the insults was some very valuable info and I thank you for that. So let me see if I get this right.
According to Ellebedy's researchers we should that "SARS-CoV-2 infection would trigger the development of BMPCs — nearly all viral infections do — but there have been signs that severe COVID-19 might disrupt the cells’ formation
2. Some early COVID-19 immunity studies also stoked worries, when they found that antibody levels plunged not long after recovery
3. "
The result of their work shows that: "As expected, SARS-CoV-2 antibodies plummeted in the four months after infection. But this decline slowed, and up to 11 months after infection, the researchers could still detect antibodies that recognized the SARS-CoV-2 spike protein. " In addition, "In 15 of the 18 bone-marrow samples, the scientists found ultra-low but detectable populations of BMPCs whose formation had been triggered by the individuals’ coronavirus infections 7–8 months before. "
To specify this a little more, there are various antibodies, against various parts of the virus, and some persist longer than others. I learned that those BMPCs keep "trickling" out antibodies inside for instance your bone marrow, possibly for the rest of your life. I assume, so correct me if I'm wrong, that in case of a infection against which they could be useful, the trickle could turn into a stream.
I learned that antibodies against the N protein have much cross-reactivity with seasonal Corona which makes them not very useful to estimate a specific attack rate. I've also learned that antibodies against the N protein are useless, and that they're "associated", or I should probably say their presence is, with higher mortality.
In looking further into T cell testing, I've also learned that it turns out there are cross-reactivity reactions in those, which would complicate, or perhaps invalidate the ability to use them as a tool for estimating attack rates.
Even after repeatedly asking what THEN would be the best way, I've been shown or taught nothing. Except perhaps, that, if the answer WAS in the paper about life-long immunity, the answer is that we DO know of a very specific antibody or more, that can be detected for life to decidedly prove a previous infection. My question is, would that involve, at least after some time, a sampling of what's in your bone marrow?
I hope you guys understand I'm serious. I want to learn and appreciate you sticking with me in this. There are too many who stop communicating.